Caffeine Intake and Dementia: Can Lewy Bodies Explain the Link?
Researchers sought to investigate the associations between habitual caffeine intake, incident cognitive impairment, and neuropathology.
Researchers sought to investigate the associations between habitual caffeine intake, incident cognitive impairment, and neuropathology.
Researchers sought to determine the effect GBA variants have on cognitive status in patients with amyotrophic lateral sclerosis.
A team of researchers investigated whether credible evidence supporting a causal relationship between Lyme disease and Alzheimer disease or Lewy body dementia exists.
A researcher assessed the predictive power of neuropsychologic features in distinguishing the progression to Alzheimer disease vs Lewy body dementia in patients with mild cognitive impairment.
Researchers explored the association between Alzheimer disease co-pathology and the presence and severity of neuropsychiatric symptoms in Lewy body dementia.
Using systematic video analysis, researchers assessed the efficacy of nelotanserin for symptomatic treatment of REM sleep behavior disorder in patients with dementia.
Patients with Lewy body dementia with likely AD-type copathology may have impaired language compared to those without such copathology.
The FDA has granted Fast Track designation to neflamapimod (EIP Pharma) for the treatment of dementia with Lewy bodies (DLB).
A number of psychiatric and neurodegenerative diseases are associated with abnormal imaging findings, which can help psychiatrists reach a diagnosis.
There were no significant differences between pimavanserin and quetiapine in time to discontinuation, mortality, or improvement in psychosis in patients with Parkinson disease and Lewy body dementia.